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November 1933

MYELITIC AND MYELOPATHIC LESIONSVI. CASES WITH MARKED CIRCULATORY INTERFERENCE AND A PICTURE OF SYRINGOMYELIA

Author Affiliations

NEW YORK

From the Neuropathological Laboratory, Montefiore Hospital.

Arch NeurPsych. 1933;30(5):1074-1085. doi:10.1001/archneurpsyc.1933.02240170126008
Abstract

Myelopathy due to extramedullary or intramedullary expanding lesions may lead to cavity formation—syringomyelia. Clinically, such cases are sometimes erroneously regarded as true syringomyelia. The myelopathic process and the secondary cavity formation may be due to: (1) destruction of tissue of the cord by an intramedullary tumor; (2) circulatory interference within the cord from compression of the intraspinal vessels by an intramedullary tumor, or (3) circulatory interference by direct compression of the cord and its vessels by thickened meninges or by an extramedullary neoplasm or by both. In some cases the pathologic process may be due to a combination of all these factors. This type of cavity formation in the cord is not to be confused with true syringomyelia, which is usually attributed to congenital developmental anomalies of the cord with dilatation of its central canal or to a central gliosis which may be due to nests of glia cells or

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