[Skip to Content]
Access to paid content on this site is currently suspended due to excessive activity being detected from your IP address Please contact the publisher to request reinstatement.
[Skip to Content Landing]
Original Contributions
June 8, 1964

Etiology of Postneonatally Acquired Cerebral Palsy

Author Affiliations

Chicago; Denver

From the Children's Neurology Service and the Hektoen Institute for Medical Research of Cook County Hospital, Chicago. Dr. Perlstein is associate professor of pediatrics at Northwestern University Medical School. Mr. Hood is associate professor and director of the Speech and Hearing Clinic of the University of Denver.

JAMA. 1964;188(10):850-854. doi:10.1001/jama.1964.03060360010002

In a retrospective study of 4,946 cerebral palsied patients, 624 (14%) had acquired the disease. This varied with the type of cerebral palsy, from 5% in athetoids to 30% in spastic hemiplegics. Encephalopathies accounted for 57% of acquired cerebral palsy, skull traumas for 18%, meningitis for 12%, vascular accidents for 8%, and all others for 5%. Sixty-five percent was acquired before the age of 2 years and 92% before 8 years. The type of cerebral palsy varies with etiology, spastic hemiplegia being most common in the vascular group. Males predominate except in the vascular accidents group. Acquired juvenile cerebral palsy is a connecting link between the congenital forms and those acquired in adult and senile life.