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JAMA 100 Years Ago
April 8, 1998


Author Affiliations

Edited by Brian P. Pace, MA, Assistant Editor.

JAMA. 1998;279(14):1126J. doi:10.1001/jama.279.14.1126

The pathologic etiology of migraine has been long enough the subject of speculation, but we are still in the phase of conjecture or at least theory; our positive acquisitions of knowledge as to the essential pathologic process are so far practically nothing. The notion that it is a vasomotor neurosis has been largely abandoned, and neurologists at the present time are inclined to class it with epilepsy as a fulgurant cortical neurosis, a symptom of cortical instability associated, it may be, with some conditions of morbid metabolism, such as the uric acid or arthritic diatheses. . . .