Of several hundred children under observation in the asthma clinic the last four years, three were encountered in whom enlarged tuberculous tracheobronchial glands produced symptoms markedly simulating asthma. This condition, judging from the literature, is comparatively infrequent in this country. Because so much attention is being focused on protein hypersensitiveness as an exciting cause of asthma, these cases are likely to be and no doubt are frequently overlooked. This condition is a definite clinical entity and of grave prognostic import. Particularly in large cities with congested immigrant settlements are children prone to develop tuberculous gland infection. According to Schick, these cases are very contagious, and it is therefore important that an early diagnosis be made, so that they can be differentiated from the noncontagious condition of true asthma.
REPORT OF CASES
—A. M., a girl, aged 2, referred to the asthma clinic, Sept. 27, 1924, was a full-term
PESHKIN MM, FINEMAN AH. ENLARGED TUBERCULOUS TRACHEOBRONCHIAL GLANDS SIMULATING ASTHMA. JAMA. 1926;86(19):1429-1432. doi:10.1001/jama.1926.02670450021009