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April 20, 1946


JAMA. 1946;130(16):1163-1164. doi:10.1001/jama.1946.02870160109012

The relation between actinic injury and carcinogenesis resulting from a therapeutic, occupational, environmental or experimental exposure to radiating energy has been inrecent years the subject of several comprehensive monographic presentations.1 The scientific importance of this problem has been enhanced by knowledge of two hazards connected with the use of atomic energy and artificially radioactive isotopes.

Lacassagne2 has recently reviewed this subject in two volumes dealing with cancers following electromagnetic (roentgen rays, gamma rays, solar and ultraviolet rays) and corpuscular (alpha rays, beta rays) radiation. He concludes that the cancerigenic action of certain radiations (roentgen rays, rays from radioactive substances, ultraviolet rays) is not the result of a chemical substance set free in the irradiated tissue under the influence of the rays but more likely a direct effect of the exposure to actinic energy of certain cellular formations controlling cell division. The diversity of rays capable of exerting a

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