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Article
October 13, 1917

THE ETIOLOGY AND DEVELOPMENT OF NEPHRITIS

Author Affiliations

SAN FRANCISCO

From the Pathological Laboratory of the Leland Stanford Junior University School of Medicine.

JAMA. 1917;LXIX(15):1223-1227. doi:10.1001/jama.1917.02590420015005
Abstract

Before entering on a discussion of my subject I wish to state that I have included in the scope of this paper only cases in which the renal tissue showed well marked diffuse hematogenous inflammatory lesions. I have excluded all focal lesions of lesser importance, whether abortive forms of true nephritis, as the embolic focal nephritis described by Löhlein1 and Baehr,2 or other focal conditions which are coincident with vascular disease and in which the disease of the blood vessels evidently constitutes the more important feature.

The cases here spoken of are those which may be designated as cases of diffuse glomerulonephritis, because the inflammatory lesions of the glomeruli play an important part in this disease and form the one connecting link in an often bewildering multiplicity of other developments. The more I study the situation both from an etiologic and a broad anatomic point of view, the

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