[Skip to Content]
Access to paid content on this site is currently suspended due to excessive activity being detected from your IP address 54.211.168.204. Please contact the publisher to request reinstatement.
[Skip to Content Landing]
Editorial
July 2005

Sweet SyndromeA Disease in Histologic Evolution?

Arch Dermatol. 2005;141(7):893-895. doi:10.1001/archderm.141.7.893

Sweet syndrome (SS) occurs in several predictable clinical settings: those with inflammatory conditions, perhaps most commonly following upper respiratory tract infection; those with hematologic dyscrasias and malignancies, most commonly involving cells of myeloid lineage; and in a variety of other settings. Sweet syndrome occurring in the setting of myeloid malignancy may histologically and clinically appear atypical for a number of reasons: clinically, some of these lesions appear purpuric owing to associated thrombocytopenia; SS occurring as a manifestation of myeloid dyscrasia does not exhibit a predominance in women, unlike the other forms of the disease; and histologically, some of these cases exhibit mononuclear cells compatible with leukemic granulocytic precursors combined with mature neutrophils.1

First Page Preview View Large
First page PDF preview
First page PDF preview
×