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Article
May 1984

Unilateral Dermatomal Superficial Telangiectasia

Author Affiliations

Dermatology Section McGuire Veterans Administration Center Richmond, VA 23249

Arch Dermatol. 1984;120(5):579-580. doi:10.1001/archderm.1984.01650410021010
Abstract

To the Editor.—  Uhlin and McCarty1 provide an additional observation compatible with the view that unilateral dermatomal superficial telangiectasia (UDST) (or unilateral nevoid telangiectasia) represents a localized increased sensitivity of the skin to female hormones. The speculation that a "dermatomal developmental defect in which estrogen receptors are increased would explain all of the situations"1(p228) in which UDST is seen, is presented without citations recognizing earlier original works that proposed this pathophysiologic mechanism based on clinical observations. In 1978, it was noted that "the unilateral dermatomal distribution of the superficial telangiectasia suggests a nevoid distribution of target end organs sensitive to estrogens."2 In this statement, credit was given to Selmanowitz3 who observed that "if a circulating hormone induced spider angiomas in the patients presented, then the unilateral distribution of the telangiectases might have represented a nevoid predisposition of target end organs in the vessels, or supportive connective

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