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Article
December 1932

DECREASED DEXTROSE TOLERANCE IN ACUTE INFECTIOUS DISEASES

Author Affiliations

CHICAGO

From the John McCormick Institute for Infectious Diseases.

Arch Intern Med (Chic). 1932;50(6):801-818. doi:10.1001/archinte.1932.00150190003001
Abstract

Transient glycosuria has been noted in various infectious diseases. Hibbard and Morrissey1 found it in diphtheria. Cammidge2 stated that it occurs in diphtheria, scarlet fever, typhoid fever, influenza, appendicitis, measles and infections with suppuration. Buhl3 described its occurrence in Asiatic cholera. Castellani and Willemore4 and Harrison5 have found that it may be present in malaria. Cammidge6 and Higginson7 have designated as "sapraemic glycosuria" that accompanying carbuncle or gangrene. This glycosuria is accompanied by a hyperglycemia according to Hollinger.8 A lowered dextrose tolerance was demonstrated by Hamman and Hirschman9 in lobar pneumonia and acute tonsillitis. Olmsted and Gay10 found abnormal blood sugar curves in many conditions including those resulting from acute infectious toxins. Tisdall, Drake and Brown11 noted a derangement of the carbohydrate metabolism in infants with acute infectious diarrhea. In the study of various infectious diseases Labbé and Boulin12 found an increase in the height of the

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