November 1950


Author Affiliations

Assistant Professor of Medicine, Northwestern University Medical School; Consultant, Division of Biology and Medicine, United States Atomic Energy Commission CHICAGO

AMA Arch Intern Med. 1950;86(5):691-710. doi:10.1001/archinte.1950.00230170044005

As a practical generalization, it may be said that sufficient irradiation of tissues with either radium or roentgen rays will cause a reduction in the number of cells of all series in the circulating blood. The greater the dose, the more profound is the blood damage, the more rapidly it develops and the more slowly it is repaired. The blood response remains quite consistent qualitatively over a wide range of dosage, although massive exposures may obscure the early changes, and small doses sometimes fail to evoke the complete response.—Minot and Spurling.1

DURING the period Aug. 6 to Dec. 16, 1945, a large number of hematological studies was performed on Japanese injured by the atomic bombs exploded over Hiroshima and Nagasaki, Japan. These studies were conducted by Japanese physicians and by medical officers of the United States Army and Navy attached to the Joint Commission for the Investigation of the

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