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February 1978

Splenic Sequestration With Sickle Cell-C Disease

Author Affiliations

From the Medical Service, West Side Veterans Administration Hospital, and the Abraham Lincoln School of Medicine, University of Illinois, Chicago.

Arch Intern Med. 1978;138(2):307-308. doi:10.1001/archinte.1978.03630260091024

During an episode of pneumonia and multiple pulmonary thromboembolic events, an adult male patient with sickle cell-C hemoglobinopathy developed a severe anemia associated with rapidly increasing splenic size and diminished splenic uptake of technetium Tc 99m sulfur colloid, indicating diminished splenic function due to hypersequestration, a rare and noteworthy phenomenon in adult patients with sickle cell-C disease.

(Arch Intern Med 138:307-308, 1978)