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March 1931


Author Affiliations

Associate Professor of Neuro-Anatomy and Neuropathology, New York University and Bellevue Hospital Medical College; Associate Neuropathologist and Adjunct Neurologist, Mount Sinai Hospital NEW YORK
From the laboratories and neurologic service of the Mount Sinai Hospital.

Arch Ophthalmol. 1931;5(3):418-444. doi:10.1001/archopht.1931.00820030102009

The quadrigeminate plate is an anatomic landmark of particular interest to the ophthalmologist. Forming the roof of the aqueduct of Sylvius (fig. 1), it overlies the narrow channel through which the cerebrospinal fluid which collects in the lateral ventricles and the third ventricle is drained into the fourth ventricle and ultimately into the subarachnoid space, where it is either reabsorbed by the pial vessels or excreted by way of the arachnoid villi (fig. 2). It is obvious that an expanding lesion in the quadrigeminate plate is in a most favorable position, by compressing this aqueduct, to obstruct partially or completely the normal flow of the cerebrospinal fluid from the ventricular compartments, rostral (anterior) to the aqueduct of Sylvius and, in this way, cause the formation of internal hydrocephalus and precipitate clinical manifestations of increased intraventricular tension. Thus an expanding lesion in this region is apt to produce, among other

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