[Skip to Content]
Access to paid content on this site is currently suspended due to excessive activity being detected from your IP address 184.73.17.193. Please contact the publisher to request reinstatement.
[Skip to Content Landing]
Article
August 1959

Ophthalmological Involvement in the Primary Demyelinating Diseases

Author Affiliations

Cincinnati

AMA Arch Ophthalmol. 1959;62(2):255-259. doi:10.1001/archopht.1959.04220020081012
Abstract

This subject has been discussed pro and Con and will continue to be an enigma until the etiology is established. Then and only in that event exacting studies and proper treatment can be instituted. At this time one does not know if the myelin destruction, frequently with subsequent damage to the axis cylinders, is from bacterial infection, toxins, poisons, or denied blood supply or whether the myelin, having a normal cycle of depletion and replenishment, i inhibited in some manner from its physiologic ability to re-form. From pathologic consideration the possibility of re-formation is hindered by the development of the glial scar or plaque and in acute cases by mesodermal infiltration.

There are certain areas confined to the central nervous system which seem to have a predilection for these demyelinating affections, and many of these areas are intimately concerned with visual and oculogyric functions, as they contain nuclei and axons

First Page Preview View Large
First page PDF preview
First page PDF preview
×