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April 1964

Tooth Ring Analysis In Cerebral Palsy

Author Affiliations

Meyer A. Perlstein, MD, 4743 N Drake Ave, Chicago, Ill 60625.; Research Scholar, and Honorary Dentist, Spastic Centre, Sydney, Australia (Dr. Watson); Professor and Head of the Department of Pedodontics, University of Illinois (Dr. Massler); Associate Professor of Pediatrics, Northwestern University Medical School (Dr. Perlstein).; From the University of Illinois, College of Dentistry.

Am J Dis Child. 1964;107(4):370-382. doi:10.1001/archpedi.1964.02080060372008

Introduction  Infantile cerebral palsy consists of syndromes characterized by chronic motor disabilities due to brain involvement, occurring primarily in the prenatal or perinatal period. It may be classified into three general subgroups1,2: (1) spastics, characterized clinically by the presence of increased stretch reflexes in the muscles and by increased deep tendon reflexes; (2) dyskinesias, characterized by abnormalities in motion without abnormal deep tendon reflexes—the most common form of dyskinesia is the athetoid; (3) ataxias, in which dyssynergia and poor balance are characteristic. The spastics and athetoids make up roughly 90% of the total cerebral palsy population.The causes of cerebral palsy are basically of two types: (1) anoxia,* which tends to be associated with brain lesions resulting in the clinical syndromes of dyskinesia, primarily athetosis, and (2) trauma and vascular damage, which tend to be associated with the clinical syndromes of spasticity. The correlation between specific syndromes and etiologies

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