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January 1927


Arch NeurPsych. 1927;17(1):44-56. doi:10.1001/archneurpsyc.1927.02200310051005

Somnolence as a symptom in cerebral lesions has been of particular interest since the recent experience with epidemic encephalitis emphasized its incidence and importance. The vast amount of literature dealing with the latter subject does not lead to definite conclusions as to the cause of somnolence, nor does the scanty literature on its occurrence in expanding lesions give more than vague and sometimes misleading information on the subject. It has been a matter of general experience to meet diagnostic problems in which somnolence has given rise to confusion, when other cerebral symptoms, such as headache, cranial nerve palsies and slight changes in the optic nerve heads, suggested epidemic encephalitis, neoplasm, meningitis and so on. Difficulties in several cases led us to investigate the subject from the standpoint of expanding intracranial neoplasms, in an attempt to understand the physical causes underlying the production of somnolence.

The occurrence of hypersomnia has long