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January 1939

SOMNOLENCE CAUSED BY HYPOTHALAMIC LESIONS IN THE MONKEY

Author Affiliations

CHICAGO

From the Institute of Neurology, Northwestern University Medical School.

Arch NeurPsych. 1939;41(1):1-23. doi:10.1001/archneurpsyc.1939.02270130011001
Abstract

Lesions have been placed in the diencephalon in 55 monkeys, and these animals have been observed for abnormalities in their reactions. A few monkeys died within a short time after the operation, and in others the lesions were not properly placed. Elimination of these unsatisfactory experiments leaves 35 animals from which valid information can be drawn. Since there are reasons for believing that either side of the hypothalamus is capable of activating structures on both sides of the body, it was necessary that the lesions should be at least approximately bilaterally symmetric. The 35 satisfactory experiments may be divided into the following groups: 11 in which the lesions involved both lateral hypothalamic areas to the level of the caudal border of the mamillary bodies; 6 in which the lesions in the lateral hypothalamic areas did not extend back beyond the rostral border of the mamillary bodies; 9 in which the

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