THE PRESUMPTION is evident that focal abnormalities of brain activity should relate to the site from which a focal seizure spreads. Much attention has been paid to the character of spread of the jacksonian seizure,1 and the accumulated evidence has been used to theorize concerning the mode of representation of movement in the motor cortex. A study of Gowers'2 excellent monograph emphasizes how much valuable data can be acquired when the details of jacksonian and other partial seizures are ascertained and the observations are related to knowledge of cerebral physiology. Indeed, Gowers' insight into the process of spread of seizures should excite the envy of the modern electrophysiologist, who will usually find that his recently discovered facts accord with Gowers' clinical deductions and rarely differ from them materially.
In examining the electroencephalograms of persons subject to focal convulsions, we were impressed with how frequently a single interseizure record