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May 1950

ANOXIC ENCEPHALOPATHY FOLLOWING POLIOMYELITIS: Report of a Case

Author Affiliations

KANSAS CITY, KAN.

From the Departments of Neurology and Pediatrics of the University of Kansas School of Medicine.

Arch NeurPsych. 1950;63(5):774-789. doi:10.1001/archneurpsyc.1950.02310230096009
Abstract

IN A RECENT study of bulbar poliomyelitis1 the conclusion was reached that many of the cerebral symptoms are secondary manifestations of anoxia2 rather than the result of direct injury to the cortex by the virus. The present paper is a report on the histologic study of a fatal case in which peculiar psychic disturbances resulted from a severe diffuse encephalopathy, ostensibly of anoxic origin.

REPORT OF CASE 

Clinical History.  —M. P., a girl aged 10 years, was admitted to the pediatric service of the University of Kansas Medical Center (Dr. Herbert Miller), on July 26, 1946. On July 23 she had complained of sore throat and began to have a fever. A local physician gave her sulfonamide drugs, but her fever continued. On July 25 she began to vomit and complained of headache and stiffness of the neck, and her temperature rose to 102 F. (38.9 C.). No

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