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May 1957

Intracerebral HematomaIts Pathology and Pathogenesis

Author Affiliations

Los Angeles

From the Cajal Laboratory of Neuropathology, Los Angeles County Hospital, Coroner's Service, Los Angeles County, and the Section of Nervous Diseases (Neuropathology), College of Medical Evangelists.

AMA Arch NeurPsych. 1957;77(5):464-472. doi:10.1001/archneurpsyc.1957.02330350030002
Abstract

Hemorrhage into the substance of the brain is one of the commonest lesions of the human body, being eclipsed, insofar as the central nervous system is concerned, only by occlusion of a major arterial channel. In the great majority of cases such hemorrhages are the result of rupture of an arteriosclerotic vessel, being precipitated by an elevated blood pressure. Other essential causes of intracerebral arterial rupture, such as trauma, embolism, ruptured aneurysm or vascular anomaly, blood dyscrasias, and syphilis, are relatively rare.1 Occasionally one encounters a case in which no cause for the hemorrhage can be found. In general, cerebral effusions may be subdivided into traumatic and "spontaneous" groups.

The onset of cerebral symptoms following hemorrhage is usually acute and immediately consequent to rupture of the diseased or injured artery. However, in many instances, particularly those consequent to trauma, evidence of hemorrhage may be delayed for hours or even

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