In my communication, "The Scaphoid Scapula, a Frequent Anomaly in Development of Hereditary, Clinical and Anatomic Significance,"1 presented to the St. Louis Medical Society, Feb. 5, 1910, I indicated the relative frequence of the scaphoid type of scapula in our population in dry bones and in fetal, embryo and monster forms. The main thesis of that communication was: The scaphoid scapula is an anomaly in development originating in the progeny from some abnormal circumstance operating in the parents, is thereafter transmitted from parent to child, and so on through several generations, and unless the abnormal circumstance again becomes operative in the descendants, the scaphoid scapula finally disappears and the racial type again becomes dominant.
I showed that it occurred most frequently in individuals deviating from the average in physical or mental endowments, or both, and stated that the natural habitat of the scaphoid scapula was in the deviate. I showed
GRAVES WW. THE CLINICAL RECOGNITION OF THE SCAPHOID TYPE OF SCAPULA AND OF SOME OF ITS CORRELATIONS. JAMA. 1910;55(1):12–17. doi:10.1001/jama.1910.04330010014006
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