[Skip to Content]
[Skip to Content Landing]
September 27, 1913


Author Affiliations


JAMA. 1913;61(13_part_2):1094-1098. doi:10.1001/jama.1913.04350140010003

Recent additions to the literature of glaucoma contribute less to its varied and many-sided clinical expression, or to the acknowledged completeness of its pathologic anatomy, than to the mystery of its origin. It is not so much the long-discussed question whether glaucoma simplex belongs in a category of its own, where the disclosures of my own experience must place it; or even that glaucoma, and in fact all its acute inflammatory expressions, are caused by blood stasis; for that a circulatory obstruction is the exciting feature has been held and believed by many investigators since Roser's time.

The more modern point of view concerns itself with the questions: (1) if in chronic, simple or non-inflammatory glaucoma, the accepted well-known sclerosis and rarefaction of the vascular and protective tissues (that is, the increased tension), is the first and actual dynamic factor and subsequently, after an inexorable but more or less uniform

First Page Preview View Large
First page PDF preview
First page PDF preview