Dust as a causative agent in the production of lung diseases has been known by writers of medicine from early times. Ramazzini, that pioneer observer of diseases of artificers, published in 1700 a treatise on the effect of dust inhalation. Stratton, in 1838, proposed the name of anthracosis to designate the condition found in coal miners as a result of inhaling coal dust, the term silicosis being applied to miners' consumption produced by quartz, flint and other silicon dusts. Probably the application of the term pneumonoconiosis, as proposed by Zenker in 1867, should be the proper nomenclature in occupational medicine for all inorganic and organic dusts that produce lung disease among workers.
The experimental work and observations of Haldane1 and of Mavrogordato2 particularly revealed a new aspect of the problem as to how pneumonoconiosis is developed, and in view of the recent investigations of the fundamental changes arising
LEGGE RT. MINERS' SILICOSIS: ITS PATHOLOGY, SYMPTOMATOLOGY AND PREVENTION. JAMA. 1923;81(10):809–810. doi:10.1001/jama.1923.02650100017006
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