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December 25, 1943


Author Affiliations


JAMA. 1943;123(17):1135-1137. doi:10.1001/jama.1943.02840520051021

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To the Editor:—  The editorial comment on the etiology of hypertension (The Journal, November 20, p. 772) discusses— and, because of its important etiologic and therapeutic implications, perhaps too briefly—a study which proposes to demonstrate that essential hypertension is of vasomotor origin. The authors (Gregory, Raymond; Lindley, E. L., and Levine, Harry: Texas Rep. Biol. & Med.1:167 [No. 2] 1943) seem to base their study on the following considerations:

  1. Essential hypertension is the result of arteriolar vasoconstriction.

  2. This vasoconstriction is either humoral or neurogenic in origin.

  3. Spinal anesthesia decreases blood pressure greatly in hypertensive subjects; in normal subjects "there was a slight fall in the blood pressure of several patients during spinal anesthesia...." However, "the blood pressures in the group with normal pressures remained essentially the same during the period of spinal anesthesia" (p. 180).

  4. It is concluded that the hypotensive effect of spinal

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