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The phenomena typical of thromballosis (and embolism), in contradistinction to apoplexy, coincident with rupture of a cerebral vessel, are of a more pronounced and complicated character than those of apoplexy.
Although apoplexy, in the aged, may be more rapidly fatal, yet an embolism may complicate with diversified phenomena, and the patient may linger for a long time.
The differentiating feature being that fatal, embolic cases the pressure upon brain tissue is from within outwards, but circumscribed by the limits of the dilated vessel, and not diffused over the adjacent tissue, similar to extra-vascular, as occurs when rupture of vessel takes place, thereby causing extensive circumjacent pressure and obliteration of cerebral function; often profound coma, or bona-fide apoplexy is caused.
We know that the predisposing cause of apoplexy is an atheromatous degeneration of the internal coat of arteries, vice-versa, in thromballosis the pathology is induced by an endarteritis or phlebitis, yet
MONETTE GN. THROMBALLOSIS. JAMA. 1884;II(20):544–545. doi:10.1001/jama.1884.02390430012001d
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