Pathologists recognize the frequency of recovery from attacks of tuberculosis of the lungs. When patients once thus affected die of other or intercurrent disease, characteristic cicatrices or calcareous condensations are found in the lungs, along with adhesion of the opposed pleura.
It would therefore seem rational to endeavor to induce or hasten such processes during the course of the disease, with the hope of destroying its cause (perhaps by preventing the nutrition of it), or at least to oppose a bar to its further progress. The posibility of such a procedure would seem all the more justifiable for the reason that the disease is and remains, in most cases, for a long time quite strictly circumscribed. It may be taken for granted that every one now believes the disease to be a mycosis. With the double purpose of directly addressing the cause of this disease with an antimycotic agent, and
WHITTAKER JT. AN ATTEMPT AT THE RADICAL TREATMENT OF TUBERCULOSIS. JAMA. 1885;IV(25):677–678. doi:10.1001/jama.1885.02391000005001a
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