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April 30, 1910


Author Affiliations

Associate Professor of Medicine (Nervous and Mental Diseases), Rush Medical College CHICAGO

JAMA. 1910;LIV(18):1427-1430. doi:10.1001/jama.1910.92550440001001d

Spastic paralysis may be either a symptom of a functional neurosis or the result of an organic lesion. If the latter, its existence indicates an involvement of the pyramidal tract somewhere between the cortex of the brain and the anterior horns of the spinal cord. It seems best to discuss first the various lesions from which spastic paralysis may result and then briefly to describe those points which are of assistance in differentiating them from the purely functional morbid states.

Spasticity and the exaggeration of deep reflexes which is associated with it are both the result of increased muscular tonus, the latter, in turn, being caused by the cutting off of the influences of regulation centers. The exact location of these centers has not been definitely determined, although there can hardly be any doubt that one of them—and that perhaps the most important one—is to be found

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