The ready applicability of modern microchemical methods to the study of various types of nitrogenous compounds other than protein that circulate in the blood has at length made it possible to throw new light on a number of long debated questions regarding metabolism.1 This now appears to be true in the case of the uric acid infarcts in the form of ammonium urates that are often found in the kidneys of the new-born during the first few days of life. That these infants excrete uric acid in unusual concentration and augmented amounts in the urine has long been known. This relatively and absolutely high output of uric acid has been correlated with associated conditions in the uric acid content of the blood by Sedgwick and Kingsbury2 at the University of Minnesota. These investigators, as well as Slemons and Bogert3 at the Yale Medical School, have found that
URIC ACID INFARCTS IN THE NEW-BORN. JAMA. 1917;LXIX(20):1699. doi:10.1001/jama.1917.02590470039014
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