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June 22, 1895


JAMA. 1895;XXIV(25):982-983. doi:10.1001/jama.1895.02430250030004

Mouth-breathing, imperfect development of the chest, anoxemia, general mal-nutrition and impaired intelligence are generally acknowledged, since the researches of W. Meyer, a quarter of a century ago, to be among the direct consequences of hypertrophy of the adenoid structure in the upper portion of the naso-pharnyx, just as truly as deafness and catarrh of the upper respiratory tract are also symptomatic of this condition. Harrison Allen, in a short communication to the Pennsylvania State Medical Society (read May 22), has still further elucidated this subject by contributing the results of his recent pathologic and clinical observations. In addition to the classical form of obstructive disorder having its site in the pharyngeal tonsil, Allen recognizes two others, each of which have interesting clinical relations which are well worth attention and investigation. The three forms of adenoid disease of the pharnyx, in accordance with this classification, therefore, will be:

  1. Hypertrophy, with mechanical