[Skip to Content]
[Skip to Content Landing]
November 9, 1895


Author Affiliations

Oculist and Aurist to St. Joseph's Hospital, Chicago; Lecturer on Ophthalmology and Otology, Chicago College of Physicians and Surgeons; President of the Alumni Association, Chicago College of Pharmacy; Member of the American Medical Association, the Chicago Academy of Medicine, etc. CHICAGO.

JAMA. 1895;XXV(19):792-793. doi:10.1001/jama.1895.02430450008001d

This article is only available in the PDF format. Download the PDF to view the article, as well as its associated figures and tables.


The frequency of eye disease originating from brain lesion is all sufficient to render an attempt to trace a closer relationship between the eyes and the brain justifiable and merits, by reason of its utility in ocular and brain diagnoses, very careful consideration. If we are dealing with structural ocular changes we are often able to define the etiologic factor at once, but at times we are confronted by functional disturbances which are but vaguely explicable; under such circumstances if we could only view the eyes as a component factor of the cerebral and cerebellar mechanism, obscurity in diagnosis would vanish. I have always regarded the eyes as nothing more nor less than prolonged bulbous expansions of the optic nerves with altered anatomic characters to meet the requisites of their physiologic necessity, viz., to formulate and receive impressions of the external world and, carrying the analogy a step further, we

First Page Preview View Large
First page PDF preview
First page PDF preview