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September 17, 1904


Author Affiliations

Instructor in Neuropathology, Harvard Medical School; Assitant in Neuropathology, Harvard Medical School, and Second Assistant Physician for Diseases of the Nervous System, Boston City Hospital. BOSTON; [From the Pathological Laboratory of the Boston City Hospital.]

JAMA. 1904;XLIII(12):789-792. doi:10.1001/jama.1904.92500120002f

The following case of hemiplegia developed in a boy during convalescence from scarlet fever. The basis of the cerebral symptoms was a markedly hemorrhage encephalitis focal in the parietal lobes. The reaction is histologicalhy characterized, aside from large areas of hemorrhage and a few superficial or meningeal areas of frank suppuration, by a tremendous proliferation of phagocytes. The phagocytes for the most part contain blood corpuscles and dilate or supplant the pial and adventitial meshes. In places, the phagocytes occupy the space of lost brain substance which has undergone acute fatty degeneration:

CLINICAL HISTORY.  G. N., an American boy of 5 years, was admitted to the South Department of the Boston City Hospital, Dec. 13, 1902, with a few hours' history of vomiting and sore throat.The boy was well developed and nourished and only moderately prostrated. The roof of the mouth was slightly reddened. The tongue was