The study of the life history of uterine fibromata, with their multiplicity of combinations, has been patient, persevering and full of vexations. At first, considered perfectly benign, we have come to recognize the probability of their producing great harm and even of taking on malignant degeneration.
Their complications are becoming constantly more numerous. Sorel1 has found they have a close relation to glycosuria. He demonstrated this relation in some marked cases of glycosuria, extending over a period of years, complicated by uterine fibroids, and in which a quick and decisive cure of the pathologic discharge of sugar followed hysterectomy.
What etiological relation, if any, exists between arteriosclerosis and uterine fibroids is still a matter of conjecture. La Force2 considers it a potent cause of fibroids of this organ, and many believe a condition of arteriosclerosis is usually present in most cases of these growths. It would seem but
BOVÉE JW. ARRHYTHMIA CORDIS COMPLICATING FIBROMATA UTERI. JAMA. 1900;XXXV(5):277–281. doi:10.1001/jama.1900.24620310011001d
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