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November 10, 1900


JAMA. 1900;XXXV(19):1207-1210. doi:10.1001/jama.1900.24620450023001g

To gather and set in orderly array the different states and conditions that have been observed and recorded as etiologic agents in the production of adenoid hypertrophy is not difficult, for the literature of the subject is extensive and at hand. Age, sex, heredity, scrofula, the lymphatic temperament, frequent colds, nasal deformities, acute infectious fevers, microbes, climate, and social condition are the chief factors that have been named. But after these have been elaborated and analyzed, we are still far from a satisfactory solution of the problem of ultimate causation in many cases. Why does it occur in one child and not in another under similar circumstances, perhaps in the same family. Why does it occur in several members of the same family, otherwise apparently healthy? What is the reason for its frequent congenital occurrence? These and other queries still clamor for solution. In the present instance we have but

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