In selecting this subject for a brief paper I do so because it is a condition that can not be discussed too often. It is only necessary to glance over the literature to satisfy one that the etiology of puerperal eclampsia is still a mooted question. Lever (from the reports of the Guy Hospital, in 1843) was the first to trace any connection between eclamptic attacks and the excretion of albuminous urine. After it became known that the urine of patients suffering from convulsions contained albumin, and they had some of the other symptoms of uremia, it was very generally considered that these patients were suffering from some form of Bright's disease. Frerichs' theory that uremia is caused from the decomposition of retained urea into carbonate of ammonia, and Traube's theory that uremia is produced in hydremic persons by a sudden increased tension of the aorta, and cerebral edema and
BEATTIE TJ. PUERPERAL ECLAMPSIA. JAMA. 1901;XXXVII(8):493–495. doi:10.1001/jama.1901.62470340011001b
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