The return of a varying degree of motor power in members paralyzed as a result of hemorrhage into the brain, occlusion of a cerebral vessel and secondary softening and other cerebral lesions has been attributed in part to the escape from destruction of certain nerve fibers, with their release from pressure and from the effects of the inflammatory process and in part to the assumption by the corresponding centers in the opposite hemisphere of the functions subserved by those of the injured side. The heightening of the reflexes and the development of contractures have in their turn been attributed to the removal of the inhibitory action of the higher over the lower centers through interruption of their channels of communication, and also to the irritation arising out of the sclerosis of the motor tracts.
For some time evidence has been accumulating that tends to throw doubt upon these explanations for
SOME REFLECTIONS ON HEMIPLEGIA. JAMA. 1902;XXXVIII(26):1691–1692. doi:10.1001/jama.1902.02480260011004
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