This article is only available in the PDF format. Download the PDF to view the article, as well as its associated figures and tables.
"R eactive hyperemia is not dependent on the perfusion pressure within the system"... nor does the myogenic response to pressure "seem to be an important factor in reactive hyperemia." These were among the observations made by Dr. Lloyd R. Yonce of the Department of Physiology at the University of North Carolina, who related results of his experiments with pressure-flow relationships in isolated gracilis muscle to physicians at the Indiana University symposium on autoregulation.
Quoting a definition set forth in 1879, Yonce said, "Reactive hyperemia is the result of 'some stimulus acting directly on the walls of the vessel or indirectly on these through the medium of nutritive changes of the tissue elements.' " More recent concepts, he added, "are no more than specific restatements of this idea." He in turn called reactive hyperemia "the manifestation of the vasodilatation or loss of tone during the period of circulatory arrest," and he offered
Reactive Hyperemia—Independent Of Blood Perfusion Pressure. JAMA. 1963;185(10):45–48. doi:https://doi.org/10.1001/jama.1963.03060100021007
Customize your JAMA Network experience by selecting one or more topics from the list below.
Create a personal account or sign in to: