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To the Editor:
—In the chairman's address before the Section on Gastro-Enterology and Proctology (The Journal, Sept. 3, 1932, p. 791), Dr. George B. Eusterman of Rochester, Minn., opened with a rapid and masterly survey of present beliefs concerning the etiology of gastric and duodenal ulcer:In summarizing these observations bearing on the etiology, pathogenesis and symptoms [said Dr. Eusterman], one cannot easily escape the conviction that nervous imbalance, resulting in disturbed parasympathetic and sympathetic interaction on gastric or duodenal motility and secretion, plays an important, if not an exclusive part, in the symptoms at least, and in the genesis of the ulcer itself. When an actual lesion does develop under such circustances, it seems justifiable to predicate a mucosal susceptibility based on inheritance, or acquired through influence of infection or other sustained irritant resulting in vulnerability of the gastric and duodenal tissue to the heightened corrosive and digestive action
Houghton HA. STATUS LYMPHATICUS RECESSIVUS. JAMA. 1932;99(22):1879–1880. doi:10.1001/jama.1932.02740740063029
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