Familial hypercholesterolemia is an important risk factor for atherosclerotic cardiovascular disease, including myocardial infarction and ischemic stroke. Familial hypercholesterolemia is a common dominant genetic condition resulting in structural, functional, and metabolic abnormalities in the low-density lipoprotein (LDL) receptor. The prevalence of the heterozygous form of familial hypercholesterolemia is reported to be 1:250 individuals, making it one of the more common genetic conditions in the US population.1