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Figure.  Association of Triple Pill Therapy With More than 50% Time at Target Blood Pressure (BP) by Subgroup (N = 700)
Association of Triple Pill Therapy With More than 50% Time at Target Blood Pressure (BP) by Subgroup (N = 700)
Table 1.  Time at Target Blood Pressure (BP) Measures, Triple Pill vs Usual Care (N = 700)
Time at Target Blood Pressure (BP) Measures, Triple Pill vs Usual Care (N = 700)
Table 2.  Mean Blood Pressure (BP) and Proportion Achieving Target BP Stratified by Time at Target (N = 700)
Mean Blood Pressure (BP) and Proportion Achieving Target BP Stratified by Time at Target (N = 700)
1.
Williams  B, Mancia  G, Spiering  W,  et al; ESC Scientific Document Group.  2018 ESC/ESH Guidelines for the management of arterial hypertension: the task force for the management of arterial hypertension of the European Society of Cardiology (ESC) and the European Society of Hypertension (ESH).   Eur Heart J. 2018;39(33):3021-3104. doi:10.1093/eurheartj/ehy339PubMedGoogle ScholarCrossref
2.
Whelton  PK, Carey  RM, Aronow  WS,  et al.  ACC/AHA/AAPA/ABC/ACPM/AGS/APhA/ASH/ASPC/NMA/PCNA guideline for the prevention, detection, evaluation, and management of high blood pressure in adults: executive summary: a report of the American College of Cardiology/American Heart Association task force on clinical practice guidelines.   J Am Coll Cardiol. 2018;71(19):2199-2269. doi:10.1016/j.jacc.2017.11.005PubMedGoogle ScholarCrossref
3.
Chung  SC, Pujades-Rodriguez  M, Duyx  B,  et al.  Time spent at blood pressure target and the risk of death and cardiovascular diseases.   PLoS One. 2018;13(9):e0202359. doi:10.1371/journal.pone.0202359PubMedGoogle ScholarCrossref
4.
Liu  K, Colangelo  LA, Daviglus  ML,  et al.  Can antihypertensive treatment restore the risk of cardiovascular disease to ideal levels?: the Coronary Artery Risk Development in Young Adults (CARDIA) study and the Multi-Ethnic Study of Atherosclerosis (MESA).   J Am Heart Assoc. 2015;4(9):e002275. doi:10.1161/JAHA.115.002275PubMedGoogle ScholarCrossref
5.
Tedla  YG, Yano  Y, Carnethon  M, Greenland  P.  Association between long-term blood pressure variability and 10-year progression in arterial stiffness: the Multiethnic Study of Atherosclerosis.   Hypertension. 2017;69(1):118-127. doi:10.1161/HYPERTENSIONAHA.116.08427PubMedGoogle ScholarCrossref
6.
Kishi  S, Teixido-Tura  G, Ning  H,  et al.  Cumulative blood pressure in early adulthood and cardiac dysfunction in middle age: the CARDIA study.   J Am Coll Cardiol. 2015;65(25):2679-2687. doi:10.1016/j.jacc.2015.04.042PubMedGoogle ScholarCrossref
7.
Kramer  H, Colangelo  L, Lewis  CE,  et al.  Cumulative exposure to systolic blood pressure during young adulthood through midlife and the urine albumin-to-creatinine ratio at midlife.   Am J Hypertens. 2017;30(5):502-509. doi:10.1093/ajh/hpx012PubMedGoogle ScholarCrossref
8.
Fatani  N, Dixon  DL, Van Tassell  BW, Fanikos  J, Buckley  LF.  Systolic blood pressure time in target range and cardiovascular outcomes in patients with hypertension.   J Am Coll Cardiol. 2021;77(10):1290-1299. doi:10.1016/j.jacc.2021.01.014PubMedGoogle ScholarCrossref
9.
Webster  R, Salam  A, de Silva  HA,  et al; TRIUMPH Study Group.  Fixed low-dose triple combination antihypertensive medication vs usual care for blood pressure control in patients with mild to moderate hypertension in sri lanka: a randomized clinical trial.   JAMA. 2018;320(6):566-579. doi:10.1001/jama.2018.10359PubMedGoogle ScholarCrossref
10.
Parati  G, Ochoa  JE, Lombardi  C, Bilo  G.  Assessment and management of blood-pressure variability.   Nat Rev Cardiol. 2013;10(3):143-155. doi:10.1038/nrcardio.2013.1PubMedGoogle ScholarCrossref
11.
Kawano  Y.  Diurnal blood pressure variation and related behavioral factors.   Hypertens Res. 2011;34(3):281-285. doi:10.1038/hr.2010.241PubMedGoogle ScholarCrossref
12.
Cuspidi  C, Ochoa  JE, Parati  G.  Seasonal variations in blood pressure: a complex phenomenon.   J Hypertens. 2012;30(7):1315-1320. doi:10.1097/HJH.0b013e328355d7f9PubMedGoogle ScholarCrossref
13.
Clement  DL, De Buyzere  ML, De Bacquer  DA,  et al; Office versus Ambulatory Pressure Study Investigators.  Prognostic value of ambulatory blood-pressure recordings in patients with treated hypertension.   N Engl J Med. 2003;348(24):2407-2415. doi:10.1056/NEJMoa022273PubMedGoogle ScholarCrossref
14.
Sega  R, Facchetti  R, Bombelli  M,  et al.  Prognostic value of ambulatory and home blood pressures compared with office blood pressure in the general population: follow-up results from the Pressioni Arteriose Monitorate e Loro Associazioni (PAMELA) study.   Circulation. 2005;111(14):1777-1783. doi:10.1161/01.CIR.0000160923.04524.5BPubMedGoogle ScholarCrossref
15.
Julius  S, Kjeldsen  SE, Weber  M,  et al; VALUE trial group.  Outcomes in hypertensive patients at high cardiovascular risk treated with regimens based on valsartan or amlodipine: the VALUE randomised trial.   Lancet. 2004;363(9426):2022-2031. doi:10.1016/S0140-6736(04)16451-9PubMedGoogle ScholarCrossref
1 Comment for this article
EXPAND ALL
pharmicokinetics
William Maliha, MD | NYS DOH, Albany Medical College: retired
In brief, at least two issues with this study:
1. No mention of what is described as "usual care". What medications were used and what were the pharmokinetics of these medications used in the usual care group.

2. All three medications in the triple drug regimen have half lives of at least 24 hours. Any or all of them would lead to higher percentages of these patients attaining target blood pressures over a full 24 hour period. Thus time of blood pressure measurement is information that needs to be supplied when comparing "usual care" to low dose triple therapy. />
An accurate assessment of "time at target" would seem to need blood pressure measurements at least twice daily preferrably 12 hours apart and correlation with the half lives of the medications used. This is especially important when one considers that many cardio-vascular events occur during sleep when the effect of many medications taken once a day are wearking off.

William Maliha, MD
CONFLICT OF INTEREST: None Reported
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Brief Report
April 13, 2022

Association of Low-Dose Triple Combination Therapy vs Usual Care With Time at Target Blood Pressure: A Secondary Analysis of the TRIUMPH Randomized Clinical Trial

Author Affiliations
  • 1The George Institute for Global Health, University of New South Wales, Sydney, New South Wales, Australia
  • 2The George Institute for Global Health, Hyderabad, India University of New South Wales, Sydney, New South Wales, Australia
  • 3Centre for Health Economics Research and Evaluation, University of Technology Sydney, Sydney, New South Wales, Australia
  • 4Clinical Trials Unit, Department of Pharmacology, Faculty of Medicine, University of Kelaniya, Kelaniya, Sri Lanka
  • 5Department of Population Medicine and Lifetsyle Diseases Prevention, Faculty of Medicine with the Division of Dentistry and Division of Medical Education in English, Medical University of Bialystok, Bialystok, Poland
  • 6All India Institute of Medical Sciences, New Delhi
  • 7Department of Epidemiology and Biostatistics, University of Auckland, Auckland, New Zealand
  • 8International Centre for Circulatory Health, National Heart and Lung Institute, Imperial College London, London, United Kingdom
  • 9Centre for Chronic Disease Control and Public Health Foundation, New Delhi, India
JAMA Cardiol. 2022;7(6):645-650. doi:10.1001/jamacardio.2022.0471
Key Points

Question  Is low-dose triple combination antihypertensive therapy associated with greater time at target blood pressure compared with usual care?

Findings  In this secondary analysis of a randomized clinical trial including patients with mild to moderate hypertension, treatment with a low-dose triple combination pill was associated with an increase in time at target compared with usual care.

Meaning  Time at target, a novel longitudinal measurement of blood pressure control, may have the capacity to augment current cross-sectional measures by being less susceptible to variability and providing more reliable and consistent measurements.

Abstract

Importance  Cumulative exposure to high blood pressure (BP) is an adverse prognostic marker. Assessments of BP control over time, such as time at target, have been developed but assessments of the effects of BP-lowering interventions on such measures are lacking.

Objective  To evaluate whether low-dose triple combination antihypertensive therapy was associated with greater rates of time at target compared with usual care.

Design, Setting, and Participants  The Triple Pill vs Usual Care Management for Patients With Mild-to-Moderate Hypertension (TRIUMPH) trial was a open-label randomized clinical trial of low-dose triple BP therapy vs usual care conducted in urban hospital clinics in Sri Lanka from February 2016 to May 2017. Adults with hypertension (systolic BP >140 mm Hg and/or diastolic BP >90 mm Hg or in patients with diabetes or chronic kidney disease, systolic BP >130 mm Hg and/or diastolic BP >80 mm Hg) requiring initiation (untreated patients) or escalation (patients receiving monotherapy) of antihypertensive therapy were included. Patients were excluded if they were currently taking 2 or more blood pressure–lowering drugs or had severe or uncontrolled blood pressure, accelerated hypertension or physician-determined need for slower titration of treatment, a contraindication to the triple combination pill therapy, an unstable medical condition, or clinically significant laboratory values deemed by researchers to be unsuitable for the study. All 700 individuals in the original trial were included in the secondary analysis. This post hoc analysis was conducted from December 2020 to December 2021.

Intervention  Once-daily fixed-dose triple combination pill (telmisartan 20 mg, amlodipine 2.5 mg, and chlorthalidone 12.5 mg) therapy vs usual care.

Main Outcomes and Measures  Between-group differences in time at target were compared over 24 weeks of follow-up, with time at target defined as percentage of time at target BP.

Results  There were a total of 700 randomized patients (mean [SD] age, 56 [11] years; 403 [57.6%] women). Patients allocated to the triple pill group (n = 349) had higher time at target compared with those in the usual care group (n = 351) over 24 weeks’ follow-up (64% vs 43%; risk difference, 21%; 95% CI, 16-26; P < .001). Almost twice as many patients receiving triple pill therapy achieved more than 50% time at target during follow-up (64% vs 37%; P < .001). The association of the triple pill with an increase in time at target was seen early, with most patients achieving more than 50% time at target by 12 weeks. Those receiving the triple pill achieved a consistently higher time at target at all follow-up periods compared with those receiving usual care (mean [SD]: 0-6 weeks, 36.3% [30.9%] vs 21.7% [28.9%]; P < .001; 6-12 weeks, 55.2% [31.9%] vs 33.7% [33.0%]; P < .001; 12-24 weeks, 66.0% [31.1%] vs 43.5% [34.3%]; P < .001).

Conclusions and Relevance  To our knowledge, this analysis provides the first estimate of time at target as an outcome assessing longitudinal BP control in a randomized clinical trial. Among patients with mild to moderate hypertension, treatment with a low-dose triple combination pill was associated with substantially higher time at target compared with usual care.

Introduction

To date, hypertension guidelines have focused on cross-sectional blood pressure (BP) targets based on 1-time measurements without consideration of the proportion of time that individuals are at target BP.1,2 Longitudinal assessments of BP control, such as time spent at target, have historically received little attention in practice. Clinical trials typically report BP control according to singular time points (often at the end of follow-up); hence there is little information on the effects of different strategies on time at target BP. Because of the high variability of BP, many people with a measure below target could have many or even most future readings above target. Observational data suggest that cumulative exposure to hypertension is an adverse prognostic marker3-7 associated with end organ damage, cardiovascular disease, and mortality.3,8

This recognition led to the concept of time at target as a new outcome for evaluating the effectiveness of BP control.3 Time at target is the percentage of time spent at target systolic and diastolic BP.3 Time at target incorporates systolic and diastolic BP, multiple time points, and BP targets in a single metric that is readily understandable; as such, it has advantages over other metrics, such as cumulative systolic BP (measured in mm HG years) or BP control rates at each visit, with multiple values per person. Chung et al3 demonstrated that higher time at target had a stronger association with a reduction in cardiovascular events than BP variability and cross-sectional BP values. A similar analysis in the Systolic Blood Pressure Intervention Trial8 also demonstrated that increased time in target systolic BP was associated with reduced cardiovascular events, but diastolic BP was not evaluated.

The strategy of initial or early use of single pill combination antihypertensive therapy may facilitate more rapid and effective BP control compared with gradual multistep titration pathways.9 However, the effect of any intervention, including single pill combinations, on time at target has not, to our knowledge, been examined, nor has this been used as an outcome in randomized clinical trials. We therefore conducted a post hoc analysis of the Triple Pill vs Usual Care Management for Patients With Mild-to-Moderate Hypertension (TRIUMPH) randomized clinical trial to evaluate whether low-dose triple combination antihypertensive pill therapy was associated with an increase time at target compared with usual care.

Methods

The details of the TRIUMPH trial9 have been reported elsewhere. In brief, the trial was conducted at 11 urban hospital outpatient departments in Sri Lanka and recruited patients with mild to moderate hypertension. Participants were randomized to a once-daily single pill combination of 3 BP lowering drugs (triple pill) at half the standard dose (telmisartan 20 mg, amlodipine 2.5 mg, and chlorthalidone 12.5 mg) or to usual care. Participants were monitored for 24 weeks. This study complied with the Declaration of Helsinki. The locally appointed ethics committee approved the research protocol and written informed consent was obtained from all participants. The study followed the Consolidated Standards of Reporting Trials (CONSORT) reporting guideline.

The primary outcome was time at target, with target BP defined as per the TRIUMPH9 protocol: systolic BP less than 140 mm Hg and/or diastolic BP less than 90 mm Hg or systolic BP less than 130 mm Hg and/or diastolic BP less than 80 mm Hg in patients with diabetes or chronic kidney disease.

Statistical Analysis

Descriptive statistics are presented as frequencies and percentages for categorical variables (eg, comorbidities and proportion of patients achieving time at target), while means and SDs are reported for continuous variables (eg, age and time at target). For each individual, time at target was calculated as the percentage of time at target systolic and diastolic BP over the follow-up period as calculated by the extrapolation method used by Chung et al3 (eFigure 1 in Supplement 1) and using BP readings at weeks 0, 6, 12, and 24. The number of days at target BP for systolic and diastolic readings were subsequently averaged. There were few missing values at each time point (<5%). An intention-to-treat analysis was used, with time at target calculated with available values. Time at target was not calculated if more than 2 BP readings were missing (n = 13).

Subgroup analyses were performed to determine mean time at target, stratified by age, sex, comorbidities, and treatment group. More than 50% time at target was chosen to identify participants achieving the majority of time at target BP. BP variability was also measured by the mean intraindividual standard deviation of follow-up systolic and diastolic BP measures in each patient (week 6, 12, and 24).10 Pearson coefficients were used to correlate BP variability and time at target. All analyses were performed using SPSS version 24.0 (IBM). All tests were 2-sided and significance was set at P < .05.

Results

Baseline patient characteristics have been described previously.9 In summary, 700 participants in the TRIUMPH trial9 were monitored for 24 weeks. The mean (SD) age was 56.2 (11.0) years, and 403 participants (57.6%) were women. A total of 220 patients (31.4%) had diabetes, 110 (15.7%) had a history of smoking, and 10 (1.4%) had a history of chronic kidney disease. Mean baseline BP was 154/90 mm Hg and 287 participants (41.0%) were receiving antihypertensive therapy prerandomization.

Table 1 shows the mean time at target and proportion of patients achieving time at target by treatment group. Participants in the triple pill group achieved a higher time at target compared with the usual care group (64.2% vs 43.4%). Patients in the triple pill group were more likely to achieve more than 50% time at target of the total follow-up time compared with the usual care group (time at target >50%-75%, 29.2% vs 21.4%; χ2, 5.38; P = .02; time at target >75%, 34.8% vs 15.1%; χ2, 35.58; P < .001) (Table 1; eFigure 2A in Supplement 1). Patients who achieved more than 50% time at target were more likely to achieve target systolic and diastolic BP at final 24-week follow-up (Table 2). One-fourth of patients spent less than 25% of time at target during follow-up, with fewer from the triple pill group compared with the usual care group (15.2% vs 34.5%; P < .001) (Table 1).

eFigure 2B in Supplement 1 demonstrates how the proportion of time spent at target changed over the course of the trial in the 0- to 6-week interval, 6- to 12-week interval, and 12- to 24-week interval. The association of time at target with triple pill therapy was seen early, with most participants achieving more than 50% time at target by 12-week study visit. Those in the triple pill group achieved a consistently higher time at target at all follow-up periods compared with those in the usual care group (mean [SD]: 0-6 weeks, 36.3% [30.9%] vs 21.7% [28.9%]; P < .001; 6-12 weeks, 55.2% [31.9%] vs 33.7% [33.0%]; P < .001; 12-24 weeks, 66.0% [31.1%] vs 43.5% [34.3%]; P < .001). Among the 300 patients who did not reach BP targets at week 6, 95 (31.7%) achieved more than 50% time at target by end of study. Among 269 patients who did not reach BP targets at week 12, 23 (8.6%) achieved more than 50% time at target by end of study.

The association between triple pill and greater time at target was consistent across subgroups, with patients in the triple pill group achieving a higher mean time at target across all subgroups over the 24-week follow-up (eTable in Supplement 1). People receiving triple pill therapy were also more likely to achieve more than 50% time at target across subgroups (Figure).

There was no significant difference in mean intraindividual systolic and diastolic BP variability between the triple pill and usual care groups during follow-up (systolic BP mean [SD]: 9.9 [7.2] mm Hg vs 10.7 [7.0] mm Hg, respectively; P = .13; diastolic BP mean [SD]: 6.2 [4.2] vs 6.3 [3.8], respectively; P = .65). However, time at target was correlated between systolic BP variability (r, −0.25; 95% CI, −0.33 to −0.18; P < .001) and diastolic BP variability (r, −0.14; 95% CI, −0.22 to −0.07; P < .001) (eFigure 3 in Supplement 1).

Discussion

In this post hoc analysis, low-dose triple combination pill therapy was associated with greater time at target compared with usual care over 24-week follow-up in patients with mild to moderate hypertension. To our knowledge, this is the first analysis of a randomized clinical trial to examine time at target as an outcome and also the first to assess the association of combination BP-lowering therapy with time at target. Treatment effects appeared to be larger than those suggested by the main trial primary outcome at 24 weeks, as time at target captured the benefits of both reduced time to BP control and more sustained BP control with low-dose triple pill therapy.

Time at target, a longitudinal measurement of BP control, may have the capacity to augment current cross-sectional measures. Current hypertension guidelines focus on cross-sectional BP targets, and BP targets are not currently expressed with a time dimension or an acceptable minimum proportion of BP measures at target.1,2 Single BP measures taken a few days or weeks apart that are often part of real-world practice are subject to poor reliability because they vary considerably by time of day11 and season.12 Whereas time at target integrating both systolic and diastolic BP at multiple time points is likely to be much less susceptible to variability and may provide more sensitive, consistent measurements compared with cross-sectional measures. Such longitudinal outcomes also provide clinically meaningful ways to detect a larger magnitude of between-group differences than identified by standard cross-sectional definitions.9

Compared with single BP measures, long-term BP load is more closely associated with hypertension-mediated organ damage and cardiovascular outcomes, as supported by 24-hour ambulatory BP monitoring and observational evidence13,14 demonstrating increased left ventricular mass index,4 incipient left ventricular dysfunction,6 and higher albuminuria.7 Compared with people with 0% time at target, patients with 25% or more time at target had one-fourth the rates of cardiovascular death, myocardial infarction, and risk of stroke.3 Even longitudinal measurements of improved time in target systolic BP are associated with reduced CV events.8 Additionally, the ability to capture reduced time to BP control may have prognostic value as a shorter time to control has been associated with lower cardiovascular risk.15 In light of this evidence, reevaluation of the current paradigm of BP targets to incorporate longitudinal targets may need to be considered.3 New BP devices, including wearable cuffless devices, may facilitate the capture of longitudinal-control at-home BP measurement and remote monitoring systems.

Limitations

Our study had several limitations. First, long-term cardiovascular outcomes were not examined, and thus the degree to which improvements in time at target translate to reduced cardiovascular events need further assessment. The trial protocol only included 4 time points for calculation of time at target. Nevertheless, this low number of visits may represent real clinical practice. The original article by Chung et al3 calculated time at target using only 1.6 BP measures per year, with time at target still correlating better with cardiovascular end points than cross-sectional end points and BP variability. Current calculations of time at target assume a linear time course of BP reduction, when in fact BP lowering effect is seen within a few weeks. Further research is needed to explore the optimal frequency of BP measurements required for time at target calculations and whether longitudinal BP assessment provides superior prediction of clinical outcomes compared with current cross-sectional definitions. Despite these limitations, this is the first trial to our knowledge to use time at target as an outcome in a randomized setting in post hoc analysis and to examine the association of a triple combination antihypertensive pill with time at target.

Conclusions

The findings suggest that among patients with mild to moderate hypertension, treatment with a triple pill containing low doses of 3 antihypertensive medications was associated with an increase in time at target compared with usual care. This study not only provides evidence that low-dose triple pill therapy may be an effective way to improve longitudinal BP control, but also introduces time at target as a novel outcome in randomized clinical trials.

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Article Information

Accepted for Publication: February 17, 2022.

Published Online: April 13, 2022. doi:10.1001/jamacardio.2022.0471

Correction: This article was corrected on June 1, 2022, to fix a typographical error in the Abstract Results section.

Corresponding Author: Anthony Rodgers, PhD, The George Institute for Global Health, University of New South Wales, Level 10, King George V Building, PO Box M201, Camperdown NSW 2050, Australia (arodgers@georgeinstitute.org).

Author Contributions: Drs Gnanenthiran and Rodgers had full access to all of the data in the study and take responsibility for the integrity of the data and the accuracy of the data analysis.

Concept and design: Gnanenthiran, Wang, Salam, Jan, Thom, Prabhakaran, Patel, Rodgers.

Acquisition, analysis, or interpretation of data: Gnanenthiran, Wang, Di Tanna, Webster, De Silva, Guggilla, Jan, Maulik, Naik, Selak, Prabhakaran, Schutte, Rodgers.

Drafting of the manuscript: Gnanenthiran, Wang, Salam, Rodgers.

Critical revision of the manuscript for important intellectual content: Gnanenthiran, Wang, Di Tanna, Webster, De Silva, Guggilla, Jan, Maulik, Naik, Selak, Thom, Prabhakaran, Schutte, Patel, Rodgers.

Statistical analysis: Gnanenthiran, Wang, Di Tanna.

Obtained funding: Webster, Guggilla, Jan, Maulik, Prabhakaran, Patel, Rodgers.

Administrative, technical, or material support: Salam, Webster, De Silva, Guggilla, Maulik, Prabhakaran.

Supervision: De Silva, Naik, Thom, Prabhakaran, Schutte, Rodgers.

Conflict of Interest Disclosures: Dr Di Tanna was previously an employee of Amgen. Dr Webster reports grants from National Health and Medical Research Council during the conduct of the study. Dr Guggilla is a shareholder of Ajanta Pharma, Asensus Surgical US, Bayer, Cellectis, Cocrystal Pharma, Divi’s Laboratories, Editas Medicine, Intercept Pharmaceuticals, Jaguar Health, Natco Pharma, Roche, and Zynerba Pharmaceuticals. Dr Maulik reports grants from National Health and Medical Research Council and the UK Research and Innovation Medical Research Council outside the submitted work. Dr Schutte reports personal fees from Servier, Novartis, Takeda, Sun Pharmaceuticals, and Abbott outside the submitted work. Dr Patel reports grants from National Health and Medical Research Council during the conduct of the study and is employed by the George Institute for Global Health, which has submitted patent applications in respect of low fixed-dose combination products for the treatment of cardiovascular or cardiometabolic disease; George Health Enterprises and its subsidiary, George Medicines, have received investment funds to develop fixed-dose combination products, including combinations of blood pressure–lowering drugs; George Health Enterprise is the social enterprise arm of the George Institute for Global Health; Dr Patel does not have a direct financial interest in these patent applications or investments. Dr Rodgers is employed by the George Institute for Global Health and Imperial College London and is seconded part-time to George Medicines, which is partly owned by George Health Enterprises, the social enterprise arm of the George Institute for Global Health; the George Institute for Global Health holds patents for ultra–low-dose fixed-dose combination products for the treatment of hypertension and diabetes, and Dr Rodgers is listed as one of the inventors (US 10,369,15; US 10,799,487; US 10,322,117; US 11,033,544); George Health Enterprises and George Medicines have received funding from public and private investors to conduct the research required for regulatory approval of cardiovascular combination products; Dr Rodgers does not have a financial interest in these patent applications or investments. No other disclosures were reported.

Funding/Support: The study was funded by grants from the Australian National Health and Medical Research Council Global Alliance for Chronic Disease (APP1041052) and the National Health and Medical Research Council (APP1052555).

Role of the Funder/Sponsor: The funders had no role in the design and conduct of the study; collection, management, analysis, and interpretation of the data; preparation, review, or approval of the manuscript; and decision to submit the manuscript for publication.

The TRIUMPH Study Group members: The members of the TRIUMPH Study Group are in Supplement 2.

References
1.
Williams  B, Mancia  G, Spiering  W,  et al; ESC Scientific Document Group.  2018 ESC/ESH Guidelines for the management of arterial hypertension: the task force for the management of arterial hypertension of the European Society of Cardiology (ESC) and the European Society of Hypertension (ESH).   Eur Heart J. 2018;39(33):3021-3104. doi:10.1093/eurheartj/ehy339PubMedGoogle ScholarCrossref
2.
Whelton  PK, Carey  RM, Aronow  WS,  et al.  ACC/AHA/AAPA/ABC/ACPM/AGS/APhA/ASH/ASPC/NMA/PCNA guideline for the prevention, detection, evaluation, and management of high blood pressure in adults: executive summary: a report of the American College of Cardiology/American Heart Association task force on clinical practice guidelines.   J Am Coll Cardiol. 2018;71(19):2199-2269. doi:10.1016/j.jacc.2017.11.005PubMedGoogle ScholarCrossref
3.
Chung  SC, Pujades-Rodriguez  M, Duyx  B,  et al.  Time spent at blood pressure target and the risk of death and cardiovascular diseases.   PLoS One. 2018;13(9):e0202359. doi:10.1371/journal.pone.0202359PubMedGoogle ScholarCrossref
4.
Liu  K, Colangelo  LA, Daviglus  ML,  et al.  Can antihypertensive treatment restore the risk of cardiovascular disease to ideal levels?: the Coronary Artery Risk Development in Young Adults (CARDIA) study and the Multi-Ethnic Study of Atherosclerosis (MESA).   J Am Heart Assoc. 2015;4(9):e002275. doi:10.1161/JAHA.115.002275PubMedGoogle ScholarCrossref
5.
Tedla  YG, Yano  Y, Carnethon  M, Greenland  P.  Association between long-term blood pressure variability and 10-year progression in arterial stiffness: the Multiethnic Study of Atherosclerosis.   Hypertension. 2017;69(1):118-127. doi:10.1161/HYPERTENSIONAHA.116.08427PubMedGoogle ScholarCrossref
6.
Kishi  S, Teixido-Tura  G, Ning  H,  et al.  Cumulative blood pressure in early adulthood and cardiac dysfunction in middle age: the CARDIA study.   J Am Coll Cardiol. 2015;65(25):2679-2687. doi:10.1016/j.jacc.2015.04.042PubMedGoogle ScholarCrossref
7.
Kramer  H, Colangelo  L, Lewis  CE,  et al.  Cumulative exposure to systolic blood pressure during young adulthood through midlife and the urine albumin-to-creatinine ratio at midlife.   Am J Hypertens. 2017;30(5):502-509. doi:10.1093/ajh/hpx012PubMedGoogle ScholarCrossref
8.
Fatani  N, Dixon  DL, Van Tassell  BW, Fanikos  J, Buckley  LF.  Systolic blood pressure time in target range and cardiovascular outcomes in patients with hypertension.   J Am Coll Cardiol. 2021;77(10):1290-1299. doi:10.1016/j.jacc.2021.01.014PubMedGoogle ScholarCrossref
9.
Webster  R, Salam  A, de Silva  HA,  et al; TRIUMPH Study Group.  Fixed low-dose triple combination antihypertensive medication vs usual care for blood pressure control in patients with mild to moderate hypertension in sri lanka: a randomized clinical trial.   JAMA. 2018;320(6):566-579. doi:10.1001/jama.2018.10359PubMedGoogle ScholarCrossref
10.
Parati  G, Ochoa  JE, Lombardi  C, Bilo  G.  Assessment and management of blood-pressure variability.   Nat Rev Cardiol. 2013;10(3):143-155. doi:10.1038/nrcardio.2013.1PubMedGoogle ScholarCrossref
11.
Kawano  Y.  Diurnal blood pressure variation and related behavioral factors.   Hypertens Res. 2011;34(3):281-285. doi:10.1038/hr.2010.241PubMedGoogle ScholarCrossref
12.
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