The etiology of cutaneous polyarteritis nodosa (PAN), a skin-limited vasculitis, is unknown.1 Hepatitis B viral infection is clearly associated with systemic PAN, but not with cutaneous PAN,2 suggesting different etiopathological mechanisms for these diseases. Recently a role for hepatitis C virus (HCV) in PAN has been debated.3-6 The aim of this study is to evaluate the prevalence of HCV infection in a cohort of patients with cutaneous PAN.