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December 1950


AMA Arch Derm Syphilol. 1950;62(6):820-828. doi:10.1001/archderm.1950.01530190054005

A REVIEW of recent standard textbooks and current literature reveals that there has been no change in thinking regarding cheilitis glandularis (cheilitis glandularis apostematosa) since the classic report of Sutton1 in the International Clinics in 1914, when he brought forth the hypothesis that cheilitis glandularis was a condition caused by a congenital hyperplasia of the mucous glands of the lips and associated mucous surfaces. Concomitant hypertrophy and hyperfunction are associated with inflammation, secondary eczematous changes and, in many cases, infection. A search of the literature, however, revealed a suggestion by Brocq2 in his ``Précis-atlas de pratique dermatologique,'' 1921, that some inflammatory diseases of the lips are manifestations of nervous instability and reference to association of the condition with hyperfunction of mucous glands of the entire buccal and pharyngeal cavity.

Recent publications on cheilitis glandularis have been almost exclusively in the European literature, and Iordan,3