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April 1987

Actinomycetoma Caused by Nocardiopsis dassonvillei

Author Affiliations

Division of Mycotic Diseases Center for Infectious Diseases Centers for Disease Control Public Health Service US Department of Health and Human Services 1600 Clifton Rd NE Atlanta, GA 30333; Dermatology Department University of Texas Medical Branch Ninth and Market streets Galveston, TX 77550

Arch Dermatol. 1987;123(4):426. doi:10.1001/archderm.1987.01660280026008

To the Editor.—  Borelli and Middleveen's letter,1 published in the October 1986 issue of the Archives, provided an opportunity to present the criteria on which the identity of the etiologic agent of the Texas case of actinomycotic mycetoma described by Sindhuphak et al2 was based.In the Centers for Disease Control's Center for Infectious Diseases, Division of Mycotic Diseases, Atlanta, the genera of the oxidative actinomycetes are differentiated from each other through the use of the well-established chemical tests developed by Hubert and Mary P. Lechevalier.3,4 Whole-cell hydrolysates of the aerobic actinomycete in question, on repeated tests and with appropriate controls, were found to contain the meso form of diaminopimelic acid. Thus, this isolate was fundamentally different from Streptomyces somaliensis and the other members of the genus Streptomyces, whose whole-cell hydrolysates contain the L form of diaminopimelic acid. Its specific identity was based on its lack of

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