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November 1954


Author Affiliations


From the University of Southern California School of Medicine, Departments of Medicine, Surgery, and Pathology, and the Los Angeles County Hospital; Assistant Clinical Professor of Medicine (Dr. Balfour), Assistant Professor of Medicine (Dr. Reynolds), Instructor in Surgery (Dr. Mikkelsen), Associate Clinical Professor of Surgery (Dr. Pattison), and Assistant Professor of Pathology (Dr. Hales).

AMA Arch Intern Med. 1954;94(5):853-858. doi:10.1001/archinte.1954.00250050167014

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THE EXHIBIT approaches the problem of portal hypertension by presenting normal and cirrhotic comparative data in the field of pathology (M. R. H.), internal medicine (D. C. B. and T. B. R.), and surgery (A. C. P. and W. P. M.). The possible physiological factors involved in causing the eleva- tion of portal pressure are demonstrated. The clinical determination of portal pressure, care of emergency bleeding, and surgical treatment as done at this institution are summarized.

Gross Vascular Anatomy of Liver.  — Figures 4, 5, and 6 illustrate vinylite corrosion casts of the vascular systems of normal, cirrhotic, and tumor-bearing livers. In the preparation of such specimens, acid-resistant colored vinylite solutions are injected into the vessels; after the plastic has solidified the surrounding tissue is digested away.In the two normal livers shown in Figure 4 there is an equal and uniform injection of the hepatic and portal venous systems.

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