Cerebral embolism is one of the three common types of cerebral vascular accident (hemorrhage, thrombosis, embolism). All are acute in onset and severely damaging in their effects, and all offer a uniformly poor prognosis. Of these three, embolism must invariably be considered as the manifestation of an underlying disease rather than as a separate primary disease process; therefore therapy must include measures intended to alleviate the primary pathological process.
The enumeration of materials which may form emboli is an unending task and includes everything from bubbles 1 to bullets.2 The following outline is representative of the variety of cerebral emboli which have been reported in the literature:
Thrombi
Septic
Bland
Fibrin
Tissue fragment emboli, originating within the victim's body
Tumors
Myxoma of heart 3
Small secondary tumor emboli
Atheromatous material
Valvular vegetations and calcifications
Marrow particles
Amniotic fluid
Foreign body emboli
Air
Fat and oil particles
Chemicals, etc.
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