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March 1985

IgA Nephropathy Associated With Disseminated Tuberculosis

Author Affiliations

From the Department of Medicine, Manhattan (NY) Veterans Administration Hospital, and the Division of Rheumatology, New York University Medical Center. Dr Cohen is currently affiliated with the Division of Hematology and Oncology, George Washington University Medical Center, Washington, DC. Dr Rosenstein was the Katherine and Gilbert Miller Fellow of the New York Arthritis Foundation and is currently on the faculty of the UMDNJ-New Jersey Medical School.

Arch Intern Med. 1985;145(3):554-556. doi:10.1001/archinte.1985.00360030206036

• A 59-year-old man had disseminated tuberculosis and microscopic hematuria, red cell casts, and normal renal function. Renal biopsy revealed focal mesangial proliferation with exclusively IgA deposits, diagnostic of IgA nephropathy. After institution of antituberculous therapy, the urinary abnormalities resolved. There is evidence to suggest that tuberculosis, in addition to other conditions associated with mucosal exposure to antigens producing an IgA immune response, can result in IgA nephropathy. This glomerulopathy is reported as a potential renal complication of concurrent mycobacterial infection.

(Arch Intern Med 1985;145:554-556)

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