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Article
July 1985

Hyperkalemic Distal Renal Tubular Acidosis and Selective Aldosterone DeficiencyCombination in a Patient With Lead Nephropathy

Arch Intern Med. 1985;145(7):1306-1307. doi:10.1001/archinte.1985.00360070188032
Abstract

• A patient with chronic renal failure, a strong history of moonshine abuse, and excessive urinary lead excretion had clinical and laboratory measurements compatible with combined hyperkalemic distal renal tubular acidosis and the syndrome of selective aldosterone deficiency. Extended treatment with fludrocortisone acetate, 0.1 to 0.2 mg/day, did not ameliorate acidosis or restore potassium excretion.

(Arch Intern Med 1985;145:1306-1307)

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