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June 1978

Hypersomnia-Sleep Apnea Due to Micrognathia: Reversal by Tracheoplasty

Author Affiliations

From the Sleep-Wake Disorders Unit, Department of Neurology (Drs Weitzman and Pollack) and Otolaryngology (Dr Borowiecki), Montefiore Hospital and Medical Center; and Albert Einstein College of Medicine Bronx, NY.

Arch Neurol. 1978;35(6):392-395. doi:10.1001/archneur.1978.00500300066013

• A 67-year-old woman with acquired micrognathia developed severe daytime hypersomnia, loud snoring, nocturnal enuresis, encopresis, and hypertension. A polysomnogram demonstrated 564 sleep apneas, primarily obstructive, recurrent hypoxia, a bradytachycardia, and absent stages III, IV, and REM sleep. Endoscopy during sleep revealed recurrent active closure of the upper pharynx associated with loud snoring. A tracheoplasty was done because of severity of symptoms and failure of conservative therapy. Dramatic improvement in sleepiness and hypertension occurred within 48 hours. On postoperative night 15 a repeated polysomnogram showed only 23 apneas, no hypoxia or bradytachycardia, and long periods of stage III, IV, and REM sleep.

Patients with the hypersomnia-sleep apnea syndrome should be provided with a tracheal opening during sleep when severe daytime somnolence, cardiac arrhythmias, and hypertension are present.

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