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July 1979

Hematomyelia With Arteriovenous Malformation of the Cord: Successful Surgical Treatment

Author Affiliations

Dept of Med Univ of Texas Health Science Center 7703 Floyd Curl Dr San Antonio, TX 78284
Dept of Neurosurg
Dept of Pediatr and Neurol Univ of Rochester School of Med Strong Memorial Hosp Rochester, NY

Arch Neurol. 1979;36(7):455. doi:10.1001/archneur.1979.00500430085026

To the Editor.—  The most common consequence of these embryologic abnormalities is gradual progression from paraparesis to paraplegia.1 Less common is the acute onset of hematomyelia, thrombosis of the malformation, or subarachnoid hemorrhage with associated deficits in function.

Report of a Case.—  A 6-year-old boy had progressive thoracic back pain of one month's duration, with an acute spastic paraparesis. Mental status, cranial nerve function, coordination of his arms and anal tone were normal. There was impairment to light touch and proprioception in the legs. Myelography showed a complete obstruction at the T-6 level due to an intramedullary mass lesion with dilated and tortuous vessels. The spinal fluid was discolored but not xanthochromic, acellular, with a protein content of 500 mg/dl and a glucose level of 52 mg/dl. At operation, distended tortuous vessels were seen between T-9 and T-12 typical of an arteriovenous malformation (AVM) of the spinal cord.At