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August 1979


Arch Neurol. 1979;36(8):500. doi:10.1001/archneur.1979.00500440070013

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Incorrect Abstract.—In the abstracts that were published in the April Archives (36:251-252, 1979), the abstract printed for Nall et al on page 251 is incorrect. The abstract should read

Acetylcholine Receptor Antibodies in Myasthenia Gravis J. Nall; P. Kornfeld; T. Mittag; A. Bender; S. H. Horowitz; A. E. Papatestas; H. Gross; G. Genkins

Dysfunction of neuromuscular transmission in myasthenia gravis is associated with a decrease in the number of acetylcholine receptors at the myoneural junction. This loss of receptors is ascribed to immunological alteration. To date, most patients with clinical myasthenia gravis have been reported to have serum antibodies to acetylcholine receptors. However, the clinical correlation with antibody titer is unclear.

The serums of 150 patients with all clinical forms of myasthenia gravis were analyzed for antibody to acetylcholine receptors. A radioimmunoassay using denervated rat muscle receptor was used. Titers varied widely within clinical groups. No correlation with sex, age

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