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July 1983

Infarction in a Meningioma After Cardiac Arrest: Computed Tomographic and Pathologic Findings

Author Affiliations

From the Departments of Neurology (Drs Pullicino, Wilbur, and Levy), Pathology (Dr Eskin), and Radiology (Dr Kido), University of Rochester (NY) School of Medicine and Dentistry.

Arch Neurol. 1983;40(7):456-457. doi:10.1001/archneur.1983.04050070086027

Tschemic degeneration in meningiomas has received little comment in the literature until recently. Penfield1 thought that it did occur and could lead to cyst formation. Cushing and Eisenhardt2 and Russell and Rubenstein,3 however, made no note of ischemie degeneration in their description of the histologie features of meningiomas.

With the introduction of computed tomographic (CT) scanning, low attenuation changes in meningiomas have been seen, not infrequently, and have been related to degenerative changes within the tumor.4·5 Russell et al5 found a 14% incidence of nonenhancing, low-attenuation areas within meningiomas. Corresponding necrotic areas with characteristics strongly suggesting ischemia were present on histologie examination, suggesting that ischemie events can occur in the natural history of meningioma.

We report the development of a low-attenuation area in a meningioma after a cardiac arrest. A postmortem examination disclosed a central area of necrosis within the tumor.


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