[Skip to Content]
[Skip to Content Landing]
Article
October 1983

Homolateral Ataxia and Crural Paresis

Author Affiliations

Palmetto General Medical Plaza Suite 401 7100 W 20th Ave Hialeah, FL 33016

Arch Neurol. 1983;40(10):659-660. doi:10.1001/archneur.1983.04050090095023
Abstract

To the Editor.  —Because of the small size of lacunae, definitive diagnosis of these isolated deep infarcts was confined to autopsy until the recent introduction of high-resolution computed tomographic (CT) scanning of the brain. Even with autopsy, good clinicopathologic correlation was not possible, owing to the time lapse between clinical ictus and death or the finding of multiple infarcts on gross examination of the brain.1 Ichikawa et al recently described a patient with a distinct clinical lacunar syndrome diagnosed by CT as "capsular ataxic hemiparesis" (Archives 1982;39: 585-586). That patient exhibited left sensory deficits and weakness sparing the face, with greater weakness in the leg than in the arm, and homolateral ataxia. There was no dysarthria. I recently observed a patient with a similar syndrome.

Report of a Case.  —A 79-year-old woman

×